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※ 학술발표대회집, 워크숍 자료집 중 4페이지 이내 논문은 '요약'만 제공되는 경우가 있으니, 구매 전에 간행물명, 페이지 수 확인 부탁 드립니다.
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원문정보
초록
영어
Muscle injuries are frequently occurred in various sports. The biological process and mechanism of muscle repair after injury are well known through the many studies. This study aimed at presenting heat shock protein and nitric oxide synthase are to respond to muscle damage and repair. This section discusses the results obtained through many arti-cles. Heat shock proteins (HSPs) are considered to play an essential role in protecting cells from damage, preparing them to survive on new environmental challenges. In addition, exercise-induced changes such as heat shock, oxidative, metabolic, muscular, and cytokine stress seem to be responsible for the HSP response to exercise. Also, inducible ni-tric oxide synthase (iNOS) generates nitric oxide (NO) for prolonged pe-riod and causes pathophysiological effects. Furthermore, iNOS is in-volved in processes such as cell injury, wound repair, embryogenesis, tissue differentiation, and suppression of tumorigenesis. In conclusion, the inhibition of HSP 70 on caspase-3 and apoptosis is associated with its inhibition on iNOS that leads to less NO production.
목차
Abstract INTRODUCTION BIOLOGICAL PROCESS OF MUSCLE REPAIR HEAT SHOCK PROTEINS NITRIC OXIDE SYNTHASE INTERACTION BETWEEN HSP 70 AND INOS CONCLUSIONS REFERENCES
키워드
Muscle injuryRepairHeat shock protein 70Inducible nitric oxide
저자
Kijeong Kim [ School of Exercise & Sport Science, College of Natural Sciences, University of Ulsan, Ulsan, Korea ]
Corresponding author